FH can result primarily from mutations in either Low Density Lipoprotein-Receptor gene (LDLR), Apolipoprotein B-100 gene (APOB), or Proprotein Convertase Subtilisin/Kexin type 9 gene (PCSK9), singly or in combination [1]. The gene discussed is VLDLR; the disease is familial hyperaldosteronism.