Figure 9B shows that in the control cells, transduced by the GFP expressing retrovirus and treated by shLuc, HCMV infection increased cellular lipid synthetic levels by greater than three fold compared to mock. PERK depletion greatly reduced the normal lipid synthetic rate in the mock infected cells and eliminated the activation by HCMV. These data agree with those shown in Figure 3B and 3C. This evidence concerns the gene EIF2AK3 and cytomegalovirus infection.