In this context, HCMV peptides might play a critical role in promoting the recognition of HLA-E by activating members of CD94/NKG2C and CD94/NKG2E receptors thus increasing susceptibility of decidual fibroblasts to dNK cell cytotoxicity at early times of infection as it has been shown previously for pNK cells [57]. The gene discussed is KLRC2; the disease is infection.