NCR1 and cytomegalovirus infection: Using Fc-chimeric proteins to block specific receptor/ligand interactions, we found that neither blockade of NKp30 (Figure 4B) nor of NKp46 (Figure 4C), both modulated upon HCMV infection, interaction with their putative ligand(s) had an effect on dNK cell killing of autologous HCMV-infected fibroblasts.