IGHG1 and neoplasm: The data listing in Table S1 shows that CAFs promote tumor cell invasion mainly via two of mechanisms: (a) altering tumor microenvironment interactions (tumor-stroma and tumor-tumor) by increasing cell adhesion (PECAM1, ITGAX, CCL4, LPP, and SIGLEC8) and cell-cell interaction signaling (CCL18, DLGAP5, C1QA, STAB1, and TEK), and (b) promoting extracellular region degradation (LYZ, C1QB, MMP9, IGLL1, and IGHG1) surrounding tumor cells (Table S3).