The data listing in Table S1 shows that CAFs promote tumor cell invasion mainly via two of mechanisms: (a) altering tumor microenvironment interactions (tumor-stroma and tumor-tumor) by increasing cell adhesion (PECAM1, ITGAX, CCL4, LPP, and SIGLEC8) and cell-cell interaction signaling (CCL18, DLGAP5, C1QA, STAB1, and TEK), and (b) promoting extracellular region degradation (LYZ, C1QB, MMP9, IGLL1, and IGHG1) surrounding tumor cells (Table S3). The gene discussed is TEK; the disease is neoplasm.