In support of our hypothesis that MBL enhances infections caused by certain glycosylated virions, there is evidence that individuals with high MBL concentrations or high-producing MBL haplotypes experience enhanced infections caused by other glycosylated intracellular pathogens such as Leishmania spp, Mycobacterium leprosum and M. tuberculosis, and opportunistic organisms in HIV-infected individuals [10], [83], [84], [85], [86]. This evidence concerns the gene MBL2 and infection.