DNM1 and infection: In addition, we found that MBL diminished the dependence on actin filaments (but not microtubules) and early endosomal acidification [70], [76] for infection (Figure 4C-F, Figure S5), suggesting that MBL may traffic viruses via a non-canonical pathway that resembles the clathrin/caveolin/dynamin/actin-independent, and microtubule-dependent endocytic pathway utilized by lymphocytic choriomeningitis virus (LCMV) and recombinant LCMV-Lassa virus GP infectious virions [77].