Similar gene expression-based analyses of the human response to bacterial infection have also revealed the importance of cytokine-cytokine receptor interactions; TLR signaling; MAPK signaling; Jak-STAT signaling; focal adhesion; and complement and coagulation cascades [63]–[65], three of which (Cytokine-cytokine receptor interactions, TLR signaling and MAPK signaling) were significantly associated with S. aureus infection in this study. Here, SOAT1 is linked to bacterial infectious disease.