In addition, AAA induced by AngII infusion is, like human AAA, more prevalent in men and positively influenced by hyperlipidemia.20 That mice lacking CD14 were protected against AAA formation induced by intra‐aortic elastase infusion and systemic AngII infusion suggests that CD14 is required for pathological processes common to both experimental models. This evidence concerns the gene CD14 and triple-A syndrome.