In addition, AAA induced by AngII infusion is, like human AAA, more prevalent in men and positively influenced by hyperlipidemia.20 That mice lacking CD14 were protected against AAA formation induced by intra‐aortic elastase infusion and systemic AngII infusion suggests that CD14 is required for pathological processes common to both experimental models. The gene discussed is CD14; the disease is hyperlipidemia.