Examination of tissue histology showed atherosclerotic changes (ie, neointimal proliferation, foam cell formation) in both AAAs and nonaneurysmal sections of aorta, whereas inflammation, medial degeneration, and oxidative stress were prominent in the AAA tissues, as has been previously reported by Miller et al.32 Aortic tissue sections were immunostained for CD14, which showed increased expression in AAA compared with non‐aneurysmal aortic tissues from the same patient (Figure 4A); immunostaining was not detected in the negative controls without primary antibody (not shown). The gene discussed is CD14; the disease is triple-A syndrome.