Several reports have suggested that IFN-γ, possibly derived from Th1 cells, can up-regulate the surface expression of the immunoinhibitory molecule B7-H1 on tumor-associated APCs (Dong et al., 2002; Liu et al., 2007; Zou and Chen, 2008; Wu et al., 2009a; Kondo et al., 2010). Here, IFNG is linked to neoplasm.