These include the ras-related tumor suppressor gene ARH1 which induces autophagy, inhibits the PI3-kinase pathway and regulates dormancy in ovarian cancer cell xenografts [4,5], the stress-activated protein kinase p38 [6], and antioxidant proteins and factors which control their expression [7] such as Mirk, which decreases the level of toxic ROS in tumor cells, increasing their survival [8] and their clonogenic growth [9]. This evidence concerns the gene DYRK1B and ovarian cancer.