Moreover, CCL20 is also involved in activating the pro-inflammatory program in thyrocytes derived from the papillary thyroid carcinoma (PTC) where the fusion protein, termed the RET/PTC1 oncogene, displays ligand-independent autophosphorylation and has been shown to activate a set of genes in vitro and in vivo[37]. The gene discussed is CCL20; the disease is differentiated thyroid carcinoma.