Several strategies are now under investigation to target Cav1 for the prevention of tumour progression and dissemination: all of them attempt to exploit the unique properties of Cav1 in molecular trafficking, and include targeting Cav1 using antisense and siRNA approaches, re-expressing ectopic caveolin, introducing a CSD and lowering cholesterol with statins or other drugs. Here, CAV1 is linked to neoplasm.