The increase in procoagulant activity after infection with all EHV-1 strains (MOI of 1 or 5) and LPS stimulation was abolished in the absence of exogenous FVIIa (Table 3, only RacL11 is shown at an MOI of 1) supporting TF-triggered, FVIIa-dependent generation of FXa. This evidence concerns the gene TF and infection.