IL22 and pulmonary fibrosis: These data indicated that CD4+ and TCRγδ+ T cells differentially expressed IL-17A and IL-22 in response to BLM treatment, suggesting that the subsets of CD4+IL-22+, TCRγδ+IL-22+, CD4+IL-17A+, and TCRγδ+IL-17A+ T cells may have distinct functions in BLM-induced pulmonary fibrosis.