3′UTR variants fitting the linkage data may contribute to differences in expression for genes including Bcap29, Dgkb, Hbp1, Pik3cg, Twistnb, and Tspan13, and, as such, remain candidates for the observed skin cancer susceptibility locus at Skts5. Etv1 and Ifrd1 remain potential candidates when considering potential epigenetic interactions or differences between susceptibility strains NIH/Ola and FVB/N at this locus [25]. This evidence concerns the gene HBP1 and skin neoplasm.