IFNG and myocarditis: Indeed, both Th1 and Th2 cells have been reported to be able to concur to myocarditis pathogenesis in EAM, although Th-1-produced IFN-γ appears to be protective (Afanasyeva et al, 2001, 2005; Cihakova & Rose, 2008; Daniels et al, 2008).