In detail, mainly elevated autoantibody levels, but also decreased titers were found against a) several heat shock protein (HSP27, HSP60, HSP70) [36], [37], [38], b) some crystallines (α-A-, α-B) [36], [38], c) structural proteins like GFAP, vimentin [38], MBP [24], d) enzymes as γ-enolase [39] and neuron specific enolase [40] or glutathione-S-transferase [41], and e) others like anti-phosphatidylserine [42], glycosaminoglycans [43], α-fodrin [30], retinaldehyde-binding protein [44] and retinal S-antigen [44] in the sera and aqueous humour of glaucoma patients. This evidence concerns the gene GFAP and glaucoma.