MYD88 and lymphoma: In the present study we found that BAY 11-7082 and BAY 11-7085 also induced the death of HBL-1 lymphoma cells expressing the MyD88[L265P] mutation, but other inhibitors of the canonical IKK complex and its activator TAK1 did not (Figure 1), which suggested that BAY 11-7082 and BAY 11-7085 were exerting their effects on HBL-1 cells by alternative/additional mechanisms and led us to investigate what the mechanism might be.