AURKA and neoplasm: Here, we found that Aur-A was high expressed in TNBC cell lines and tumor samples, and inhibition of Aur-A kinase by VX-680 or RNAi can efficiently inhibit cell proliferation and decrease cell migration in TNBC cells (Figure 5, Figure S3), suggesting that inhibition of Aur-A kinase may be a potential therapeutic strategy for TNBC treatment.