Overall, D518 essentially resists against BPH attack via increased activity of proteins related to metabolism (Glyoxalase I, Probable ATP synthase 24 kDa subunit, Enolase), stress response (S-like RNAse, GSH-dependent dehydro ascorbate reductase, Salt stress root protein “RS1”) and protein synthesis (Chloroplast translation elongation factor Tu1) (Table 3). Here, RS1 is linked to benign prostatic hyperplasia.