Disorganization in the extracellular matrix modulation has also been attributed to the pterygium formation[14,15].Pterygium epithelial cells (PEC) and limbal epithelial cells (LEC), but not normal conjunctival epithelial cells (CEC), respond to UVB exposure by an enhanced production of metalloproteinase-1 (MMP-1), an enzyme involved in the turnover of extracellular matrix, supporting the concept of UV-induced genetic trauma to LEC as a pathogenetic mechanism for pterygium[14]. Here, MMP1 is linked to pterygium.