Autoantibodies with strong clinical correlation to neurological disease in SLE are the aPL (observed in our study in 8/16 patients), the anti-ribosomal P antibodies (2/16 patients), and the NMO-IgG/anti-AQP4 antibodies that appeared in 2 out of 4 patients with myelopathy [10], [31], [47]. The gene discussed is FASLG; the disease is neuromyelitis optica.