Lung adenocarcinoma (LADC), the most common form of non-small-cell lung cancer (NSCLC), comprises several different genomic subsets defined by unique oncogenic alterations, and a considerable proportion of LADC cases harbor driver alterations in the EGFR, KRAS and ALK genes at the mutually exclusive manner with rare exceptions [2]–[5]. This evidence concerns the gene EGFR and non-small cell lung carcinoma.