HCMV infection was previously shown to augment angiogenesis and lymphangiogenesis [21], to induce cancer cell motility, invasion and adhesion to endothelial cells [50], [51], to stimulate NF-κB signaling pathways [40] and to promote resistance of cancer cells to chemotherapy [52], all of which are also properties that characterize IBC disease [33], [39], [53], [54]. This evidence concerns the gene NFKB1 and cytomegalovirus infection.