It should be noted that the proteins involved in the metabolic myopathies glycogen storage disease type 13 (GSD13) and glycogen storage disease type 5 (GSD5), that are β-enolase (ENO3) and the muscle form of glycogen phosphorylase (PYGM), respectively, present a high number of physical interactions with the LGMD proteins group. Here, ENO3 is linked to glycogen storage disease due to muscle beta-enolase deficiency.