Fabry disease (FD; Online mendelian inheritance in man (OMIM) #301500) is a X-linked (Xq22.1) inborn error of glycosphingolipid catabolism resulting from deficient α-galactosidase A activity (GLA; OMIM #300644) due to mutations in the GLA coding region, leading to the systemic accumulation of globotriaoslyceramide (Gb3 or GL-3) in the plasma and cellular lysosomes of vessels, nerves, tissues, and organs [4]. Here, GLA is linked to Fabry disease.