ATAD2 and breast carcinoma: Trichostatin-A inhibited growth in the same cell lines which were dependent on MYC both in the endometrial (R2 = 0.74, p = 0.013; Figure 4e), and in the breast cancer cell lines (R2 = 0.31, p = 0.007; Figure 4f) but the overall efficacy of Trichostatin-A at reducing cell viability was lower among the doses we tested (0.04–10 μM) (Table S9) than were the effects of MYC or ATAD2 knockdown.