In untreated cells, HSV-1 BAC Luc virus infection resulted in ∼5000000 units of luciferase activity, and gBs treatment caused a dramatic drop in luciferase activity to ∼1000000 units (Fig. 1C), suggesting that gBs binds to cell surface receptors and inhibits HSV-1 BAC Luc virus entry. The gene discussed is CD177; the disease is viral infectious disease.