Therefore it is not unreasonable todeduce that CsA recipients who carry p.Leu101 variants of FPR1 may be more susceptible topathogen infection than those displaying the p.Val101 phenotype, and this is especiallynoteworthy in a population with a relatively high p.Leu101 allele frequency such as theHan Chinese. The gene discussed is FPR1; the disease is infection.