(Bueno et al. (2008) found that miR-203 functions as a tumor suppressor andis silenced by hypermethylation in hematopoietic malignancies expressing either ABL1 orBCR/ABL1.Chaubey et al. (2009) detected thatmiR-219-2 and miR-199b can be hemizygously lost in a significant proportion of CML caseswith der (9q) deletion. Here, ABL1 is linked to neoplasm.