Altered expression of four genes, ATPase, Na+/K+ transporting, beta 1 polypeptide (Atp1b1), Growth hormone (Gh), acidic leucine-rich nuclear phosphoprotein 32 family member A (Anp32a) and Granulin (Grn) at the very early time of post-infection provided insights into the mechanism of pathogenesis [61]. This evidence concerns the gene ANP32A and infection.