Regarding potential targeted therapies for ovarian cancer patients in the future, it is important to consider that PARP1 was reported not only to promote base excision repair and microhomology-mediated NHEJ but also to play a role in the detection of stalled replication forks, namely, in the recruitment of the MRN complex for end processing, and also in homologous recombination to restart the stalled replication fork [62]. This evidence concerns the gene PARP1 and ovarian carcinoma.