When free testosterone and insulin replaced PCOS status, ucOC was predicted only by β-CrossLaps (p = 0.0002) and cFT (p = 0.003) in the model (F = 13.710, p < 0.001), meaning that 46.71% of serum ucOC variability was explained by these two parameters. The gene discussed is INS; the disease is polycystic ovary syndrome.