FBXO32 and hypertrophic cardiomyopathy: The present results showed that many genes significantly altered in cell development and hypertrophic cardiomyopathy by overexpressed atrogin-1, Myh6, Serca2 (ATP2A2) and Prkcz were significantly up-regulated, whereas Itga3 (also known as CD49c) and Foxa2 were down-regulated.(Table 4).