Although the precise mechanisms of cancer cachexia are not fully elucidated, it is now clear that proinflammatory cytokines, such as interleukin-6 (IL-6) and tumor necrosis factor-α (TNF-α), which are derived from both tumor cells or host cells, are involved in induction and development of cancer cachexia [4, 5]. This evidence concerns the gene IL6 and cancer.