Among those cases, Deng et al. reported in their case that HAML showed atypical angiomatous, epithelioid components with pleomorphic and frequent mitosis in the center of the large tumor displayed p53 immunoreactivity, and mutation at exon 7 for p53 and resulted in vascular invasion, distant metastasis, and fatal outcomes [23]. The gene discussed is TP53; the disease is neoplasm.