Since dysregulation of Wnt/β-catenin signaling pathway that leads to uncontrolled tumor cell proliferation, metastasis, and resistance to apoptosis has been reported in lung cancer [48, 49], Wnt and its downstream targets, GSK-3β, β-catenin, and MMPs, were included to study the effects of MCME on CL1 cells. This evidence concerns the gene GSK3B and neoplasm.