We expected the K14-HPV-E7 mice to have some level of dysplasia due to the expression of the E7 oncogene, as well as the wild-type mice to experience some mild cell changes as a result of the chlamydial infection, but we did not expect both wild-type and transgenic infected mice to develop CIN II. This evidence concerns the gene KRT14 and chlamydia trachomatis infectious disease.