As reviewed by Blackwell et al. [55], several polymorphisms that facilitate uncontrolled inflammatory responses, in particular those resulting in the underproduction of the anti-inflammatory cytokine interleukin-10 (IL-10) or the overexpression of the proinflammatory cytokines IL-1β and IL-6, may be found at a higher proportion in SIDS victims than in controls. This evidence concerns the gene IL10 and sudden infant death syndrome.