Wang et al. [24] reported that Lp(a) and ox-Lp(a) concentrations in active RA were higher than those in both inactive RA and control; Lp(a)-immune complex (IC) concentrations in active RA were also higher than inactive RA, while no difference was found in Lp(a), ox-Lp(a), and Lp(a)-IC concentrations between inactive RA and controls. Here, LPA is linked to rheumatoid arthritis.