There were no significant differences in serum activin A, activin B, follistatin, and HsCRP between the normal, impaired glucose tolerance (IFG/IGT), and T2D groups in the subject population (Table 1), although there was clear evidence of a trend towards an increase in both activins and follistatin in the T2D group compared with the other two groups. This evidence concerns the gene INHBE and Impaired glucose tolerance.