Indeed, a cytokine able to increase the adhesive properties of the endothelial wall [21, 22], and that it is implicated in the pathology associated with experimental uveitis [23], the TNF-alpha was drastically reduced following the treatment of the rats with RvD1 as well as the chemokine CXCL8, a chemokine able to recruit neutrophils in rodent species during experimental inflammation [21, 24]. The gene discussed is TNF; the disease is uveitis.