Following these interesting results, a more recent paper showed blockade of CCL2 following influenza infection led to an increase in epithelial damage [34], with this being explained by the reduced numbers of cells leaving the AEC more open to infection, ascribing a protective role only to AM, and indeed a role in repairing the influenza-infected epithelium following clearance of the infection. The gene discussed is CCL2; the disease is influenza.