LMO1 and acute lymphoblastic leukemia: These alternative 5′ start sites coincide with peaks of non-coding sequence conservation suggesting possible roles as alternative promoters for LMO1. This is compatible with the work done previously,20, 24 where two alternative promoters of LMO1 were proposed following northern hybridisation in the LMO1-translocated T-ALL cell line RPMI8402 and the human neuroectodermal small cell lung cancer-derived cell line N417.