We observed that, counter-intuitively, only resident CD11b+Ly6C− DCs were able to transfer airway allergy to naïve recipient mice (Figure 6a–c) and to promote type 2 cytokine, IL-17 and, to a lesser extent, IFN-γ production from BLN cells (Figure 6d–e). Here, IL17A is linked to allergic respiratory disease.