In vitro studies indicated dysbindin as a binding partner of several proteins with a suggested role in muscular physiology[24-26], and increased dysbindin transcript and protein levels were measured in muscle biopsies from individuals and mice with Duchenne Muscular Dystrophy (DMD; MIM 310200) [27,28]. The gene discussed is DTNBP1; the disease is Duchenne muscular dystrophy.