IL17A and asthma: Consistent with the observed inhibition of cells infiltrating the lungs, we also found that exposure to ES-62 suppressed IL-17 responses, which have recently been shown to play key roles in recruitment of eosinophils, neutrophils and macrophages as well as in the promotion of Th2 polarised inflammation and development of severe asthma (Lajoie et al., 2010; Wang et al., 2010; Iwakura et al., 2011; Barin et al., 2012; Dias and Banerjee, 2012).