GAST and alkaline phosphatase measurement: The outcomes of the study provide harmonic coherence of gene-chip analysis and biochemical assay data using samples from the animal model, suggesting a novel mechanism that the onset of AGML is, at least partly, due to the gastrin, and gastric acid / somatostain imbalance triggered by the toxins in the AP serum; and cannabinoid agonist HU210 restores the equilibrium, hence the protection.