IP-10 expression can be highly induced mainly by IFN-γ but also by other cytokines like IL-2, IFN-α, IFN-β, IL-27, IL-17, IL-23, TNF-α and IL-1b.IP-10 is considered to function as a chemoattractor for monocytes and T cells at inflammatory foci [8], and it has been observed that its plasma level increased in patients with ATB and significantly reduced upon successful TB treatment, which could be useful in monitoring the disease activity and efficacy of therapy [9], [10]. Here, IFNG is linked to tuberculosis.