The expression of produced and/or acquired TF by neutrophils is an attractive and realistic scenario for the pathogenesis of thrombotic events that characterize several inflammatory disorders, including sepsis (Aras et al., 2004), ANCA-associated vasculitis and Behçet's disease (Tomasson et al., 2009), or inflammatory bowel disease (Miehsler et al., 2004). This evidence concerns the gene TF and Sepsis.