The oncogenic potential of EBV is mainly due to the expression of the LMP1 viral oncogene that functionally mimics CD40 signal activation and elicits powerful proliferative stimuli in the target cells [2], [37] Recently, De Leo et al. reported that resveratrol inhibits the proliferation and survival of EBV-infected Burkitt’s lymphoma cells [40]. The gene discussed is PDLIM7; the disease is Burkitt lymphoma.